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Ified variants are12 characterised in an in vitro system, which lacks physiological context. Therefore, creating a zebrafish equivalent of the human LQT7 mutations and assessing their characteristics in zebrafish may complement cell culture-based studies. The assessment of dysmorphic features, muscle defects, and if possible cardiac phenotypes would add value to the characterisation of unclassifie
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Expression of myocardiumspecific structural genes (connexin 43, troponin T, and HCN4 ion channel) (Formigli et al., 2009). Coronary infusion of C2C12 skeletal myoblasts overexpressing relaxin in conjunction with administration of exogenous relaxin starting at day 30 after infarction in rats improves myocardial viability in the infarcted area compared with myoblast therapy alone (Bonacchi et al., 2
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